Noise Pollution Research Paper

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Noise pollution is undesired sound that is disruptive or dangerous and can cause harm to life, nature, and property. It is often said that noise differs from other forms of pollution in that, unlike atmospheric pollutants for example, once abated, noise leaves no residual accumulation in the environment or the human  body. Noise does leave behind its effects, however, and these can deteriorate after continued exposure to harmful sounds. So it is not true, strictly speaking, that “noise … leaves no visible evidence” (Lai 1996, p. 389).

The hazardous effects of noise depend on its intensity (loudness in decibels), duration, and frequency (high or low). High and low pitch is more damaging than middle frequencies, and white noise covering the entire frequency spectrum is less harmful than noise of a specific pitch. Noise may be ambient (constantly present in the background) or peak (shorter, louder sounds).

Noise-induced hearing loss (NIHL) in humans is the major, though by no means only, problem stemming from noise pollution. In 1978 the U.S. Environmental Protection Agency (EPA) Office of Noise Abatement and Control estimated that around twenty million Americans were exposed daily to noise resulting in permanent hearing loss (EPA 1978). In 1990 about thirty million people in the United States were exposed daily to occupational noise levels above 85 decibels, compared with just over 9 million people in 1981. Exposure for more than 8 hours a day to sound in excess of 85 decibels is potentially hazardous. In  Germany and other developed countries, as many as four to five million people, that is, 12 to 15 percent of all employed people, are exposed to noise levels of 85 decibels or more (World Health Organization 2001).

Loud, abrupt sounds can harm the eardrum, while sustained sounds at lower volume can damage the middle ear; both types of sounds can cause psychological damage. Noise disrupts sleep and communication, and numerous studies have documented  the  heart-related, respiratory, neurological, and  other  physiological effects of  noise. Stress, high blood pressure, anger and frustration, lower resistance to disease and infection, circulatory problems, ulcers, asthma, colitis, headaches, gastrointestinal disorders, and  many  other  physiological and  psychological problems have been linked directly to noise. In addition, children have been shown to suffer from slower language development and  disruption  of learning as a result of noise. More  than  five million children  in  the  United States, ages six to  nineteen, suffer from noise-induced hearing impairment (Havas 2006). In the United Kingdom,  Netherlands,  and  Spain,  exposure to  noise impaired children’s reading comprehension and caused a delay in reading skills development (Clark and Stansfeld 2005). In Austria, children in noisier neighborhoods were shown to  suffer from  increased stress and  diminished motivation (Evans et al. 2001). A fetus exposed to noise may experience a change in heart rate, or it may suffer the impact of its mother’s noise-related stress.

In addition, noise can harm animals and the environment, as well as physical property. Livestock and pets are harmed by noise, as are animals in the wild. Noise can also disturb wildlife feeding and breeding. Noise-related property damage includes structural damage from vibrations induced by sound waves and economic harm in the form of lower property values. The true social costs of noise pollution also must include monetary losses from sickness, absenteeism, loss of productivity and  earning capacity, and much more.

Noise pollution is not new, but it has become more problematic with the developments associated with industrialization and urbanization. Between 1987 and 1997, community noise levels in the United  States were estimated to have increased by 11 percent and were predicted to continue increasing at that rate or more (Staples 1997). Commercial and  industrial activities, construction,  aircraft, vehicular traffic (highway and off-road), and the rapid increase in the use of machines and other technologies are all associated with noise pollution. Modern household appliances and lawn and gardening equipment are increasingly common sources of noise. Like many other forms of pollution, noise appears to disproportionately affect poor and disadvantaged minority communities, and so is also an environmental justice issue.

In the United States, public policy to address noise pollution began in the early 1970s. The Noise Control Act of 1972 charged the federal government with protecting public health and welfare from noise pollution  by establishing standards for noise emissions and by authorizing federal agencies to establish rules. The EPA created the Office of Noise Abatement and Control (ONAC) as a result of the Noise Control Act. The Quiet Communities Act of 1978 authorized the EPA to provide grants to state and local governments for noise abatement. In the early 1980s the Occupational Safety and Health Administration (OSHA) set standards for industrial noise exposure and criteria for hearing protection. The OSHA guidelines resulted in a reduction of noise levels and hearing loss to workers, but some hearing loss can occur even at OSHAapproved levels. In 1981 Congress agreed to the Ronald Reagan administration’s  proposal to  cease funding  for ONAC,  although  Congress did  not  repeal the  Noise Control Act when it eliminated ONAC’s funding.

Noise pollution can be controlled through reduction at the source, interruption of transmission paths, or protection of the receiver. Reengineering machines and simply turning down volume when possible are methods of reduction at the source. Barriers, enclosures, and other forms of soundproofing can interrupt transmission paths. The use of hearing protection is the main form of receiver protection. Experts recommend a multifaceted approach, including appropriate training on the use of equipment and on why ear protection matters, enforcement of hearing-protection regulations, and the use of new technologies that reduce noise at the source (Lusk et al. 2004). Like many  other  environmental problems, addressing noise pollution is complicated by issues of shared responsibility and  jurisdiction, making some conventional economic approaches less effective and inviting new interdisciplinary solutions. New active noise control (ANC) technologies may assist in dealing with noise pollution in the years ahead through the use of digital processors that convert analog sounds into  digital signals, allowing computergenerated “antinoise” to erase sound with sound (Alper 1991).

While market-based approaches to pollution control have become more popular in recent years, there have not yet  been  any  emissions trading  or  pollution  permits schemes applied to noise. It should be recalled, however, that up until the time of the first government regulation of pollution, a market-based approach was the “default” mode of pollution control.

Bibliograhy:

  1. Alper, J 1991. Antinoise Creates the Sounds of Silence. Science 252 (5005): 508–509.
  2. Clark, Charlotte, and Stephen Stansfeld. 2005. The Effect of Aircraft and Road Traffic Noise on Children’s Reading. Literacy Today 44 (9): 24–25.
  3. Evans, Gary W., et al. Community Noise Exposure and Stress in Children. Journal of the Acoustical Society of America
  4. 109 (3): 1023–1027.
  5. Havas, V 2006. Noise! The Invisible Pollution. Current Health 2 32 (5): 10–11.
  6. Lai, P 1996. Noise Pollution. In Major Environmental Issues Facing the 21st Century, eds. Mary K. Theodore and Louis Theodore, 389–396. Upper Saddle River, NJ: Prentice Hall.
  7. Lusk, Sally, et al. Acute Effects of Noise on Blood Pressure and Heart Rate. Archives of Environmental Health 59 (8):
  8. 392–399.
  9. Staples, Susan 1997. Public Policy and Environmental Noise: Modeling Exposure or Understanding Effects. American Journal of Public Health 87 (12): 2063–2067.
  10. S. Environmental Protection Agency, Office of Noise Abatement and Control. 1978. Noise: A Health Problem. http://www.nonoise.org/library/epahlth/epahlth.htm.
  11. World Health O 2001. Fact Sheet No. 258. Geneva: WHO Press. http://www.who.int/mediacentre/factsheets/ fs258/en/.

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